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Autistic Pride Day

2 Jun

This month, on the 18th is something called Autistic Pride Day. It probably won’t make the news, you almost certainly won’t see a large march and you won’t have to put up with hearing z-list celebs talk about their solidarity and respect for the celebrants.

Autistic Pride Day is a day that adult autistics chose to commemorate the fact of their autism and the fact that they were proud to be autistic. It should not be confused with Autism Awareness which is a campaign set up and managed mostly by parents and carers of autistics to highlight what they see as the tragic blight of autism.

Autistics today face an incredible amount of generalisation and pejoratives. I’ve heard other parents of autistics describe their own children as – ‘disappearing down a black hole’, ‘stolen’, ‘missing’, ‘a parents worst nightmare’ and their own family situations described in terms reflecting grief and tragedy.

Once upon a time, homosexuality was considered an illness. In fact it wasn’t until 1973 that the American Psychiatric Association (APA) removed homosexuality from its official listing of mental illnesses in its Diagnostic and Statistical Manual of Psychiatric Disorders.

1973. Just over 30 years ago. It seems incredible that people really did think that a persons sexual preference was symptomatic of an illness, but they did. Up until that point, gay Americans were subjected to pejoratives and mind-sets very similar to those that autistics face now. The ‘deviance from normality’ was too much for most families so a lot of gays stayed quiet or grew estranged from their families. Autistic people don’t even have the option of staying quiet. Their ‘deviance from the norm’ leads them to ‘stim’ (flap hands, rock, sing to themselves and a massive variety of other ‘deviant’ behaviours). Its obvious to everyone when an autistic is autistic.

The Black man, it was repeatedly claimed, was uniquely fitted for bondage by his primitive psychological organization. For him, mental health was contentment with his subservient lot, while protest was an infallible symptom of derangement. Thus a well-known physician of the ante-bellum South, Dr. Samuel Cartwright of Louisiana, had a psychiatric explanation for runaway slaves. He diagnosed their malady as drapetomania, literally the flight-from-home-madness, ‘as much a disease of the mind as any other species of mental alienation.’ Another ailment peculiar to Black people was dysaethesia Aethiopica, sometimes called rascality by overseers, but actually due to ‘ insensibility of nerves’ and ‘hebetude of mind’, explained Dr. Cartwright. Whereas psychologically normal Negroes were faithful and happy –go-lucky, the mentally afflicted ones ‘pay no attention to the rights of property…slight their work…raise disturbances with their overseers.

Scientific racism.

The shocking quote above, demonstrates how people one believed that black people were considered ill. How a Doctor could believe that a person desiring freedom could be construed as an illness is bizarre. But it happened.

Benjamin Rush, MD (1746 – ­1813), signer of the Declaration of Independence, Dean of the Medical School at the University of Pennsylvania and the “Father of American Psychiatry, “described Negroes as suffering from an affliction called Negritude, which was thought to be a mild form of leprosy. The only cure for the disorder was to become white.

Vanessa Jackson.

If such a hypothesis was even suggested today, the author would be quite rightly hounded out of the medical profession. Its utterly ridiculous. But it happened.

Autistics today face similar situations, similar ill-informed judgments and similar results. A lot of autistics say that growing up they felt forced to try and bury their autism as much as possible to avoid the judgment of family and peers. Would we find a gay teen having to do this acceptable today? Whats the consensus of opinion on Micheal Jacksons continuing quest to make himself white?

In the days leading up to Autistic Pride day, I’m going to try and write something every day about why I feel this is such an important event for Megan and for our whole family. I’m also going to try and link to articles that offer some insight into what its like to be on the receiving end of prejudice. The first article is written by Amanda Baggs and describes how the mainstream autism advocacy movement (by which I mean medics and parents mostly) are participating in the belittling of autistics.

As to what we’ll be doing on Autistic Pride Day, we’re not sure. My wife’s due to give birth to our new baby sometime this month so it may be on or around this very day. But if it isn’t, then we’ll be passing it quietly with Megan. Doing things that she likes to do. We’ll also be thinking of all the autistic adults and children we know and wishing them well and hoping that equality for them comes sooner than it did for gay people or black people.

Star Wars III: Superfluous Opinion

1 Jun

It says something about me that I don’t want to think about too much that straight after watching Episode III I wanted to blog my reactions. Does this make me sad for being a web geek or Star Wars geek? Actually, don’t answer that.

The film can be sectioned in three. Section One: Mostly OK. Section Two: So bad it made me want to stab my eyes out. Section Three: Mostly good.

Section one was full of typical Lucas over-reliance on SFX. One scene featured General Grievous’ ship approaching a planet, then landing on the planet, then GG himself leaving the ship and walking off whereupon we switch to another scene. What the hell was the point of this scene?. Some of the effects were really good – the top-down view of the space battle looking toward the planet surface was great – but George, please – use ’em sparingly!

Section two. Oh dear. Has no one told Haydn that he can’t act? This section concentrates on building up the relationship between Anakin and Padme and it…is…dreadful. The writing is just appalling:

ANAKIN: You are so beautiful!

PADME: It’s only because I’m so in love . . .

ANAKIN: No, it’s because I’m so in love with you.

PADME: So love has blinded you?

ANAKIN: Well, that’s not exactly what I meant . . .

PADME: But it’s probably true!

They laugh.

What??? What does that even mean?

Section three is an improvement. This really starts when Sidius kills all the Jedi (from invincible to inept in 5 seconds – so much for the Force). Haydn still can’t act but at least he can glower convincingly. The ending fight between Anakin and Kenobi is really good and some (gasp!) decent acting from Ewan McGregor allows us to feel Kenobi’s pain at maiming his apprentice. Not before some more terrible dialogue though. At the end of the fight Kenobi jumps into firm ground – Anakin moves to come after him:

OBI-WAN: It’s over, Anakin. I have the high ground.

Hmm. If only ever General in the history of warfare had realised that merely being slightly higher than your opponent meant you’d automatically win any fight. Bet they feel silly now.

The creation of Darth Vader was excellent but they should’ve left it when he started doing the patented Darth heavy breathing, as subsequent scenes saw Vader perfecting his Al Jolson routine (Stiff legs, hand waggling) in a vastly amusing manner. Not what you want to see from the Lord of Darkness.

Still a good film just don’t go expecting Shakespeare.

The Flash RSS Reader

30 May

A few people have expressed curiosity in it so I thought I’d run through how its done. At this stage its really very simple but I have plans to extend its operation to automate more of the running of it.

Macromedia first introduced the ability for the SWF format to parse XML data back in the days of Flash 5 (2000? 2001?). It was pretty basic was pretty shoddily implemented as well. It was never quite resolved whether it was an issue with the actual actionscript events or the Flash Player failing to parse these events (my money was on the latter) but suffice it to say, whilst it was great to be able to introduce another area of flexibility to SWF, it wasn’t until the release of Flash MX and the version 7 player that SWF/XML integration became a more reliable process.

It was around that time that Actionscript Guru, Sam Wan released his RSSDataFactory object which made coding the integration of RSS feeds very easy. He claims it only works with RSS 0.92 but it works fine with RSS 2.0 also.

So, I turned to this RSS object to handle the laborious work of processing the RSS. It was originally built to handle just one feed so I had a few tasks to make it operational for multiple feeds. I also had the thorny issue of the infamous Flash Sandbox to negotiate. More on that later.

Customising the RSSDataFactory is a doddle. First of all I simply inserted another keyframe and moved the DataFactory code to the second keyframe and applied a universal stop(); action to all keyframes. So, now I had two frames to play in I could put all my top level links in frame one and the parsed RSS feeds would display on frame two. Easy.

In frame one then I simply listed all the feeds I was going to be utilising as buttons and coded them appropriately.

Now you may notice at this stage that I’m not calling the feed directly. This is due to the pernicious influence of the Flash Sandbox. The Sandbox is a security measure. It doesn’t allow movies or scripts from servers on domains other than the one the SWF exists on to be accessed directly from the SWF. So what we have to do is build a sort of proxy – a script that _can_ access remote files and, as this proxy script exists on our domain, we can access anything it returns. Problem solved. Now you can see why calling remote XML files is pointless – nothing would be returned due to the Sandbox – so instead I call the proxy script. And because I have multiple feeds, I append a name/value pair (....php?feed=1) to each separate button. I can then detect these in my proxy script and hence return the desired script:

Now we turn our attention to this proxy script (which I called ‘antisandbox.php’). What we’re doing here is detecting the incoming value of the variable ‘feed’ fetching the appropriate RSS feed and opening it up for our SWF to process.

There are a few ways to do this, notably using readfile() but they are mostly (and again, notably readfile()) badly insecure. The way to securely get the feeds is to use (in PHP anyway) CURL. CURL opens a decent, protocoled connection to the remote RSS script. Its syntactically very like using a session in that you initialise it, use it then close it:

I should really have used switch to do the evaluation of the feed variable as its quicker. Probably will implement that soon.

So now we’ve got our data, we have to parse it in. Thankfully all this is done in Sam’s excellent Factory. All I had to do was pass it the value of the proxy script with the right name/value pair.

The last line of this script

rssObj.parseFeed(_root.theFeed, this, "receiveRSSObject");

calls the SWF variable I set in the previous frame that specified which value to send to the proxy script (and thus which RSS feed to fetch).

These are quite interesting:

In fame two I built a movieclip template. This template contains an empty dynamic text holder and an empty URL variable. When the RSS feed comes in, depending on how many posts each author specified their feeds help, each one of these posts would duplicate the movieclip and send the title of each post in the feed and the correct permanent link to these posts. It also spaces out each newly generated movieclip below the previously generated one.

My last problem was the ‘back’ button. Going back a frame in Flash doesn’t affect whats in the frame you’ve just left so I had to clear these newly generated movieclips, clear theFeed and reset the whole shebang everytime someone clicked the ‘back’ button.

Note I simply hide the ‘old’ buttons off stage – this speeds up regeneration if a user wants to revisit a certain feed in the same session.

And thats it for now. I will be extending this to make my life easier. For now, if I want to add a new feed I have to edit the FLA and ‘antisandbox.php’ which is a bit of a pain. What I hope to do is write a plug-in for WordPress that will create an XML file from a link category in the WordPress database architecture. I can then use this XML file to auto generate the feed list in frame 1. The second half of this plug in will drag these details from the database and execute them in ‘antisandbox.php’.

Accessibility – Know Your Audience?

26 May

Read the recent ‘A Conceptual Framework for Accessibility Tools to Benefit Users with Cognitive Disabilities‘ report from WebAIM, its clear that there’s one overriding concern that has to be addressed before we can successfuly design websites for users with cognitive disabilities. We need to know our audience.

As web designers we (I hope!) perform extensive usability testing with differing Scenarios. I wonder how much of that testing concentrates on accessibility? When we develop our Personas do we cater for our markets and forget that some in these markets are disabled? Some even are cognitively disabled. I’m willing to bet that we don’t. In fact I’m willing to bet that lots of web designers don’t even build Scenarios let alone build Personas representing disabled members of the target markets to move through those Scenarios.

Its easier for the majority of web designers to not bother. And for some web designers who make the laudable decision to make their sites accessible, there’s a belief that simply adhering to Priority 1 (A) of the WCAG means they’ve done all they need to. I know I’m guilty of this on occassion.

However laudable and useful doing this is or isn’t, its still not addressing the key point – we, as web designers, don’t make enough effort to know this sub-group of our audience. We read articles telling us to validate to WCAG standards and we do it but that should not absolve us from getting to know our audience better. The use of Personas and Scenarios has given us a massively useful way of getting to know key users but they are based on aspects of ourselves or people that we know as well as aset of marketing data. The last time you built a set of Personas how many of them had Downs Syndrome? How many were wheelchair bound? I’ll be honest – I have the advantage of having an autistic daughter and thus have an insight into that condition and ways in which I can account for that group of users but 9 times out of 10 my Personas are based solely on marketing data – ages, gender, income, likes, political beliefs, religious beliefs.

Why is this? We’ve all taken on board the figures – the disabled community have a vast amount of spending power – but can we really say we feel comfortable at knowing how we meet these users needs. No, we can’t. Why? Because we don’t know them. Our liberal upbringings state that we need to see the people, not the disability and for all aspects of social interaction I totally agree. But when it comes to building something that works then we need to see the people and the disability. We need to be curious – how does someone with Downs Syndrome percieve an icon? How does someone with Dyslexia read content? What thought processes lead an autistic to make a choice from navigation options?

We won’t know until we ask. Its not rude to ask. Its not rude to be curious. Its rude to assume. I’m not suggesting that you patronisingly go out and befriend everyone you can find with some kind of cognitive disability but what I am suggesting is next time you go to the effort of building Personas for usability testing, try and include someone with autism, or someone with ADD, or a generic learning difficulty. And don’t make the disability an appendage, make it what it is – an integral part of who that person is. If you need (as I do) to learn more about cognitive disabilities then don’t research it on the internet, don’t chat to people on a forum – include these people in your focus groups, get to know them and your Personas will become more accurate as a result.

1 In 166. Really?

26 May

In a recent blog post, David Kirby repeated the oft-cited figure of 1 in 166 people now having autism. That figure, according to Kirby, used to be 1 in 5000 in the eighties.

He goes on to ask that if autism is solely genetic then where are all the old autistics now?

As a writer, Kirby is pretty good. As a researcher he’s suspect and prone to errors. As a critical thinker he’s woeful.

Something happened between now and the eighties alright. Something that can definitely account for the increase in prevelance. Simply put – the criteria changed. What constituted autism changed between the eighties and now. Changed drastically. Changed so much in fact that a lot of diagnosticians doubt that Hans Asperger would recognise what is now considered to define Aspergers Syndrome, changed so much that people who were once thought of as AS would be today diagnosed as autistic.

The diagnostic criteria in the DSM, which provide a differentiation between autism and Asperger’s syndrome, have been examined by several research studies over the last five years. There has been some criticism from clinicians and research that the criteria do not identify the disorder Hans Asperger originally described. The four cases he described in his original paper would be diagnosed, according to DSM criteria, as having autism not Asperger’s syndrome. (Miller and Ozonoff 1997).

Dr Tony Attwood

So here we are with this sudden reclassification of a lot of peoples diagnosis. Further, those who would one have been classed as having Aspergers are now routinely being diagnosed as being autistic. I don’t have any stats (and I doubt they exist) on these numbers but common sense would indicate that these people make a real and significant (and continuing) impact on prevelance rates.

Going back one stage for a moment – who exactly is Kirby referring to with this ‘1 in 166’ figure? Is he talking about the prevelance of ASD? Or is he talking about the prevelance of Kanners (classical) autism? You see, the words ‘autism’ and ‘ASD’ are, in modern medical history, interchangeable. I don’t know but my gut feeling is that when people quote this 1 in 166 number they are referring to ASD – the spectrum of autism and not Kanners autism in particular.

If I’m right then we have another huge amount of people to add to our prevelance rates – the recently diagnosed. As stated the diagnostic criteria has significantly widened and as it has, the number of medical and non-medical people trained to spot ASD’s has risen massively. It doesn’t take a genius to figure out what would happen if you vastly widened the qualifying criteria for something and vastly increased the opportunity to see if people did qualify.

There are people (I know some of them) who in their thirties suddenly discover that the problems they’ve managed to live with since childhood actually have a name and an overridding condition. These people get diagnosed too.

Why is it that I know about these people and the likes of Kirby and the various mercury zealots do not (or choose not to?). Because I talk to autistic people. I don’t talk to their parents, or their carers or their advocates. I talk to them. I hear their stories and it all comes out – some (most) were diagnosed in childhood but a significant (and growing) number are getting their diagnosis’ as adults. They can do this because more is known about autism and their are more places to go and ask. When you couple this with the widening of the diagnostic criteria then you suddenly include a whole range of middle aged people who weren’t able to be included in ‘official’ stats before.

So can these people account for all of the ‘1 in 166’. I have no idea. But then I also have no idea who Kirby et al are actually counting when they reach these figures. Kanners autistics? Those with Aspgers Syndrome? Those with a generalised Pervasive Developmental Disorder? Tourettes? In the eighties it was Kanners. Today, autism is all of the above.

Variation in the administrative prevalence of ASD is associated with education-related spending, which may be associated with better-trained educational staff who can recognize the problem, and more and better trained in-school specialists who can provide screening. It is also associated with the availability of health care resources. Increased access to pediatricians and school-based health centers may lead to improved recognition of ASD. Interstate variability in the identification of ASD should be taken into account when interpreting the results of prevalence studies based on administrative data and the associated system characteristics taken into account by policy makers working to improve the recognition of ASD.

David S. Mandell, ScD; Raymond Palmer, PhD

The incidence of research-identified autism increased in Olmsted County from 1976 to 1997, with the increase occurring among young children after the introduction of broader, more precise diagnostic criteria, increased availability of services, and increased awareness of autism. Although it is possible that unidentified environmental factors have contributed to an increase in autism, the timing of the increase suggests that it may be due to improved awareness, changes in diagnostic criteria, and availability of services, leading to identification of previously unrecognized young children with autism.

William J. Barbaresi, MD; Slavica K. Katusic, MD; Robert C. Colligan, PhD; Amy L. Weaver, MS; Steven J. Jacobsen, MD, PhD

We observed dramatic increases in the prevalence of autism spectrum disorder as a primary special educational disability starting in the 1991-1992 school year, and the trends show no sign of abatement. We found no corresponding decrease in any special educational disability category to suggest diagnostic substitution as an explanation for the autism trends in Minnesota. We could not assess changes in actual disease incidence with these data, but federal and state administrative changes in policy and law favoring better identification and reporting of autism are likely contributing factors to the prevalence increases and may imply that autism spectrum disorder has been underdiagnosed in the past.

James G. Gurney, PhD; Melissa S. Fritz, MPH; Kirsten K. Ness, MPH; Phillip Sievers, MA; Craig J. Newschaffer, PhD; Elsa G. Shapiro, PhD

SEO And Dissemination Of Google PR

21 May

I read a great post from Cody regarding SEO tips recently. I don’t agree with every point – in my experience it makes no difference how far up the code block your content is for example – and there’s a couple he missed – notably the importance of submitting your site to the major directories – but its still a great list.

Something thats rarely touched on outside of dedicated SEO communities is the best way to get a good Google PR structure for your site. Its not as simple as get a good PR on the home page. In fact sometimes that can be counter-productive.

What Is PR?

Google’s PageRank (PR) technology is a mark of importance that Google attach to a particular page. Allegedly. There is a significant school of thought that claim PR is useless as a measurement of the success of a site in Google and yet another school of thought think that it was once used seriously by the Google search algorithm but since the Autumn 2003 ‘Florida’ event, Google have been using it less and less and some even suspect its used as a double-blind technique to make SEO black-hatters (those who use dodgy SEO techniques) falsely optomise their pages for PR.

Personally, I think its all about site structure and that PR isn’t so much part of Google’s seach algorithm but more a mark of how well you are doing _in_ that algorithm.

The Trick

Not really a trick – nothing ever is in good SEO – but more a long hard slog to get the right balance. Its very difficult originally but once you get the idea behind it and put in the donkey work you start to see some real results.

Think of a web forum – any web forum, the subject is irrelevant – look at the structure of it. How a web forum builds its structure should be how you are aiming to get a good site balance that will give you a good, well disseminated PR thats passed through to every page of your site.

A forum is tiered. Every seperate tier ‘drills down’ to a more concentrated and specialised area of the forum. Lets take a web forum about cookery for example. Teir one (or level one if you prefer that terminology) is simply ‘cookery’.

Tier two breaks that down into slightly more targetted areas – say, ‘utensils’, ‘cooks’ and ‘recipies’ for tier two headings.

Tier three goes into even greater targetted areas. Tier three of the second tier about ‘recipies’ for example might be ‘french recipies’, ‘spanish recipies’, ‘italian recipies’ etc etc you get the idea. Tier four of each of these tier three areas are your ‘money’ pages.

Lets go back to tier one and demonstrate how we use keywords throughout these levels. On tier one, the keywords are very generic and concentrate _solely_ on the idea of cookery. You don’t make _any mention_ of the keywords you’ll be using on tiers two/three/four/etc (depending on how ‘deep’ your site structure is). Everything on tier one is fairly generic regarding cookery.

Tier two is again fairly generic about the seperate categories you have here and your keywords should make brief mention of tier one but should be weighted for keywords regarding tier two but again, make no mention of any tiers _below_ this level.

Tier three’s keywords make very brief reference to tier one, a decent reference to tier two but are very specific to tier three. Once more – no mention of any tiers below this one should be made.

OK, I’ve designated tier four as our ‘money’ pages – this is as far as my hypothetical cookery site content actually drills to. So this page can mention tier one in passing, make brief reference to tier two, brief reference to tier three but really go to town on this (its own) tier. This is where you start heavy (but not too heavy) mentions of your three and four keyword phrases. Make your pages as readable as possible for humans – _all_ humans – by which I mean make your site as accessible as you can. An accessible site always ranks better as it is readble by both humans and machines alike. If your pages are easily viewable and navigable with styles turned off (or in Lynx) then you’re definitely on the right tracks.

So the goal with your keywords/phrases is to make them more and more specific on each level. Tier one concentrates on a very broad subject and tier four deals with very specific areas within that subject. Tier one should concentrate on one, possibly two generic keywords and tier four should make use of four or five very targetted two/three word keyphrases.

Doing this properly should result (eventually) in a PR of 2, 3 or 4 on your home page and as you get more relevant anywhere between 6-8 on your heavily targetted tier four pages. These tier four pages are where you should aim to get all your backlinks pointing to (a backlink is exactly as it sounds – a link back to you. Please note: a backlink is _not_ the same as a link exchange which won’t get you as good rewards). Depending on your market (and some markets can take a very long time) if your tier four pages get a lot of high quality backlinks then your PR on these pages can get up to the 8-9 level.

Another thing to note – the PR level in the Google toolbar frequently differs from the real PR. For a more accurate picture of your PR, try and find your site in the Google directory. If its not there you need to submit your site to DMOZ as soon as possible as it can take a *long* time to get a site accepted in DMOZ. A site I submitted in Jan 2004 still isn’t listed today for example. Obviously this entirely depends on how busy your category is but don’t leave this standing – sort it out as soon as you can.

Web Designers And Autism

21 May

I read a post at Matthew’s blog today describing his current feelings towards his own motivation in which he said:

I think a lot of ‘creative’ types would agree that it is very easy to self-diagnose with borderline manic depression, OCD, Aspergers, and a variety of other personality traits that go with the territory of trying to create something that other people will appreciate…

Now obviously with my daughter being classicaly autistic and Aspergers being a companion to Kanners autism (they are both autistic spectrum disorders – ASD’s) my interest was immediately piqued.

I commented that I had noted that a _lot_ of my fellow designers seemed, from reading their comments or blog postings, to have definite ASD characteristics – most notably of Aspergers Syndrome – but I then chickened out and refused to name names.

It seemed only fair though to offer my fellow designers a chance to find out for themselves whether they display a disposition towards autism and to that end I’ll point you to the online ‘AQ’ (autistic quotient) test.

Unlike a lot of online tests, this one won’t tell you what sort of Star Wars character you are or what your Hobbit name is. This one measures your responses to key questions that determine whether or not you have a disposition towards autism. Its developed by a team at Cambridge University led by Professor Simon Baron-Cohen (if you recognise that name then yes, they are related – its his cousin).

If you score 32 or higher then you have an increased chance of having a disposition towards autism. Its nothing to worry unduly about – if you’ve coped fine up till now then there’s no reason why you shouldn’t continue to do so but if you do struggle and you get this score or more then maybe you should see your Doctor.

I’m also happy to tell you more about autism and point you to some great resources if you need it. Add a comment or email me privately if you’d rather.

NB: The CGI script thats processing the results seems to be broken – manual scoring is at the bottom of the test.

Kirby Posts On The Huffington Blog

21 May

David Kirby posts one of what he considers to be the key questions:

There is no such thing as a genetic epidemic. Yet U.S. autism rates have gone from about 1-in-5000 in the 1980s to 1-in-166 today. But if autism is purely genetic (without an environmental “trigger”) and has always been prevalent at the same constant rate, then where are the 1-in-166 autistic 25-year-olds (those born in 1980)? Where are the 1-in-166 autistic 55-year-olds? Why can’t we find them?

Kirby.

NB: I don’t think autism is solely genetic. I think all genuine autistics have a genetic predisposition but I also think that for some, an environmental trigger event could be required. That trigger though is definitely not vaccines.

I don’t have an easy answer to this one – because data collection on autism is historically very bad its difficult to even verify Kirby’s figures. I do have a few suggestions for him though.

First, please quotes your sources when you make assertions like this. I’ve already shown you to be factually wrong on figures before. Forgive me if I don’t take everything you say at face value.

Secondly, as you probably well know, the diagnostic criteria for ASD vastly altered in the 9o’s to include a wide variety of PDD’s including for example, Aspergers Syndrome. I don’t have any figures to hand to state what the exact before/exact after was but common sense dictates that a large part of this so-called ‘epidemic’ is down to this. There would also seem to be at least one book thatdoes have the figures and they deduce the same as me. I’ll get a hold of that as soon as I can.

Thirdly, detection methods and centres of detection have vastly bettered and increased respectively.

I know it is not a very popular view, but it is possible that the dramatic increase of children with autism world-wide is due to better diagnosis, better awareness, changing diagnosis, and the growth of services. When I started in this field 20 years ago, there were only about 3 centres in the whole of the UK where one could go to get one’s child diagnosed with autism, from a specialist. Today, there are many clinics in every town that can provide this service.

Professor Simon Baron-Cohen

Another question for Kirby: If autism is mercury poisoning why is it that boys make up three quarters of all autistics? Are we to believe Thimerosal is selective?

As a point of note to sensationalist reporters turned writers – apply Occams Razor first: look for what is most likely based on the facts at hand. If the facts can’t account for everything, then perform an investigation. Your basic error is that you would seem to have skipped the first stage of that process.

Imaginary Conversations Part II

19 May

[phone rings]

Me: ‘lo

Client: Hey Kev its me.

Me: Hey Generic Client, how’s it going?

Client: Not bad, not bad. I just wanted to ring to let you know we’re all really loving the work you’re doing for us. We’re all pretty excited about how it’s going.

Me: Nice one Generic Client – remember though, don’t clap, just throw money! AHAHAHAHA.

Client: Sorry?

Me: It was a joke dude. Forget it. Jesus.

Client: Right. Anyway, as I say, we’re all simply adoring your input on this one. Seriously, we love it. There’s just one tiny problem we need you to sort out.

Me: OK.

Client: Little flags.

Me: Little flags?

Client: Yeah, you know – to indicate the different languages our clients might speak. We basically want 10 different versions of the site. Each sperate version should be written in a seperate language – Spanish, French, German, American…

Me:…and on the home page you want a flag for each of these guys that they click to access ‘their’ site. Am I right?

Client: Totally! We knew you’d see the potential. And make them animated – like they’re in a breeze.

Me: A breeze.

Client: Yeah!

Me: But don’t you remember Generic Client? We talked about this – I said I’d write a script that would detect the incoming IP of each visitor, perform a reverse look up on it, resolve the country of origin for that visitor and swap all the content in based on that persons native language.

Client: Oh. Right.

Me: We agreed it would be easier guy – ‘one design, many contents’ remember? Makes your life much easier when it comes to updating the design or structure.

Client: I guess.

Me: So whats with the grief?

Client: We really like the little flag idea. I mean *really* like it. We had a board meeting and everything.

Me: I’m telling you Generic Client, having 10 versions of your site is such a bad idea that it could well be the worst idea that anyone’s ever had. Ever. In the history of ideas. In fact, This idea makes Captain Oates idea of ‘going for a little walk’ look like one of the stellar ideas of the world. When the lookout on the Titanic decided to look the other way for a bit, that was, it can’t be denied, something of an erroneous decision. But its not as bad as this. I’m begging you Generic Client – don’t do this.

Client: OK, OK! We’ll stick with your idea…..

Me: Nice one.

Client…..but…..

Me: What?

Client: Is there some way we can use the little flag thing? It’d look so cool!

Me: I’ll see what I can do. Christ.

Client: You rock! Did I tell you how much we love you over here?

[phone down]

Evidence Of Harm States Its case

17 May

David Kirby unveils the extent of his evidence that mercury causes autism and presents it in the ever eager to please Schafer Autism Report.

Very recent studies, presented by top university researchers at major autism conferences or published in respected, peer-reviewed journals, have reported the following findings possibly supporting a link between mercury and autism. Some of this work has been derided by the CDC as ‘junk science’ conducted by ‘charlatans’.

So lets get rid of the dross and spin first. “major autism conferences” as a source is frankly, laughable. Not only are they unnamed, they are likely to be run by hysterical parent-led groups. I think its safe to discount these as credible. This leaves us with ‘top university researchers’ publishing in ‘respected, peer-reviewed journals’. Lets also bear in mind that that, by Kirby’s own admission these are ‘findings’ (as oppose to evidence or facts) that ‘possibly’ (as oppose to do) ‘support’ (as oppose to are) a link (as oppose to cause) autism.

So lets write out what Kirby and his motley crew wish they could say here and what they can say:

Kirby Crew: “Autism is mercury poisoning – here is some factual evidence to back that up.”

Reality: “There may be a link to indicate the possibility that some forms of mercury might have some relationship to autistic-like symptoms.”

There is not one shred of evidence that I know of that would prove that mercury poisoning causes autism.

Univeristy Of Washington / National Institutes Of Environmental Health Sciences – Published In Environmental Health Perspectives. In primates, ethylmercury from vaccines (in the form of thimerosal), once it enters the brain, converts to inorganic mercury at two to three times the rate of methylmercury, which is found in fish. Inorganic mercury lacks a natural transport system out of the brain, where it remains for long periods of time, perhaps indefinitely. A previous study by the same team found that inorganic mercury was the main cause of serious changes in brain tissue, including a major expansion of microglial cells (white matter), which is consistent with the finding of “big brains” in autistic children.

Note firstly the lack of a direct quote from the article itself. Once you get past the spin you can only deduce from the above that primates brains changed when you put mercury into them. Apparently this is ‘consistent’ with autistics big brains.

At this point I’d like to reveal the results of my own scientific investigation. Once, I gave an ice cream to a monkey which it promptly ate and shrieked in a monkey-like way in gratitude. As autistic children eat ice cream and occassionaly make strange shrieking noises too I hereby annouce that ice-cream causes autism. Where’s my Nobel prize?

In all seriousness, no one is saying that putting Mercury into kids in elevated quantities is a good thing. Its right there should be limits and maybe even a ban. This is because it can lead to mercury poisoning. Not because it causes autism. There really is a difference.

University Of California – M.I.N.D. Institute – Presented At The 2005 Interational Meeting For Autism Research. Children with autism have a markedly different immune profile from normal kids. They are found to have increased autoimmunity, extremely high levels of certain immune cells and cytokines, and an imbalance of immune cells to antibodies (TH1 vs. TH2 response). ALL of these abnormal conditions appear in the literature on mercury toxicity.

What? How is this evidence? All it shows is that some aspects of some autistics immune profile share some characteristics of mercury toxicity. I don’t know anyone in the field who doesn’t know that autism and mercury poisoning share characteristics. How exactly does this even support a link, let alone offer supporting evidence?

Johns Hopkins Bloomberg School Of Public Health – Published In The Journal Pediatrics. The rate of increase in new cases of autism among children born every year in the United States was relatively stable until 1987, when the rate suddenly began to spike, and then continued to rise among children born in each subsequent birth cohort. A second spike in the rate of increase was noted in 1992, a few years after which, the rate began to level off. (It is interesting to note that, between 1987 and 1992, with the introduction of new thimerosal containing vaccines, total mercury exposure from infant immunization went from 75 to nearly 240 micrograms). Meanwhile, the reported incidence of mental retardation and other childhood disorders remained constant, meaning that “diagnostic substitution” was not an explanation for the rise in autism cases.

Demonstrating both a frightening attitude to misinformation and a nice line in arrogance regarding how the US sees itself as the centre of the world. Firstly, what happens in America is relevant to America only. The rest of us don’t see that tally. If Kirby wants to rename his book ‘Evidence of Harm Between the Years 1987-1992 and Only In America’ based on this ‘evidence’ then that might go some way towards addressing his interpretation of data. Still won’t make it any more relevant though. And ‘diagnostic substitution’? What the hell? Is Kirby suggesting that Doctors don’t know the difference betwe autism and mental retardation? Or is this just a really stupid way of trying to persuade people the the autism ‘epidemic’ isn’t better detection and reporting plus an increase in the width of diagnostic criteria? I suspect the latter.

University Of Arkansas – Arkansas Children’s Hospital Published In The Journal Biology. Children with autism have extremely low or depleted levels of sulfur-based bio-chemicals known as thiols (a synonym for thiol is mercaptan, or literally, “mercury capturer,”). The reason is thought to be genetic. Without these substances, such as the protein glutathione, these genetically variant children suffer from oxidative stress, and show a reduced capacity to liminate heavy metals like mercury. Biomedical intervention with a variety of natural substances was shown to elevate thiol levels in study children to normal levels.

Er, OK. So what Kirby seems to be saying here is that if autistics have low levels of thiols they can’t liminate mercury. How that supports the theory of mercury poisoning causing autism is beyond me. This line of argument is almost a living breathing definition of the word ‘circumstantial’. Or ‘crap’.

Columbia University – Published In Molecular Psychology. Mice with a genetic predisposition to autoimmunity showed horrific reactions to thimerosal containing vaccines, compared to mice without the autoimmunity. Sensitive mice showed repetitive and self-injurious behavior, including grooming themselves or their cage mates incessantly, sometimes to the point of death. They also were found to have increased brain size compared to the typical (control) mice exposed to the same vaccine schedule.

I must admit to the odd snigger or two by this point. Is this really what Kirby thinks is ‘supporting evidence’ of mercury as a causative of autism? Tell you what Dave mate – when I see two autistics grooming each other to the point of death then I’ll start to worry.

Here’s what Craig Newschaffer of Johns Hopkins says:

Epidemiologist Craig Newschaffer of Johns Hopkins says that animal experiments such as this are important to determine the physiological effects of exposure to toxic substances. But, he notes, it’s impossible to say with certainty that lab animals exhibiting certain kinds of behavior have autism or that what happens in lab animals translates to people.

Science News.

Of course, the mercury crowd don’t care for logic. How they think you can diagnose a mouse with autism is simply baffling.

Northeastern University – Published In Molecular Psychiatry. Thimerosal, when exposed to cells with certain genetic mutations, can interfere with critical metabolic processes, including something called methylation. Methylation is crucial for proper gene expression and DNA/RNA growth, and for the development of neurotransmitters and essential fatty acids – including myelin – which protect the nerves and brain. Methylation is also needed for the development of sulfur-based thiols (or “mercury capturers”) such as glutathione, and other detoxification functions.

So really, what you’re saying is that if someone doesn’t have these ‘certain genetic mutations’ then Thimerosal can’t harm them? But…but…I thought all the mercury crowd thought there was no genetic basis for autism. Talking to such hysterics as Lujene Clarke gave me the idea that it was either/or with you guys. And even if you do support the idea of a genetic basis – what exactly are these ‘certain genetic mutations’? I can’t help but note no-one says what these mutations actually are. Possibly because they have no bearing on autism?

Here’s what proper scientisits have to say about this study:

The authors conclude that “The potent inhibition of this pathway by ethanol, lead, mercury, aluminum and thimerosal suggests that it may be an important target of neurodevelopmental toxins.” Unfortunately, the authors tacked on the statement that “…vaccine components (i.e. thimerosal and aluminum) may have contributed to the risk of autism, ADHD and other developmental disorders.” We understand the pressures on scientists to justify the relevance of their research and maintain funding of their work but the problem is that sometimes basic research is just basic research and nothing more.

Medicinenet

Next!

University Of Texas – Published In The Journal Health And Place. Mercury released primarily from coal-fired power plants may be contributing to an increase in the number of cases of autism. The study found that autism increased in Texas counties as mercury emissions rose. For every thousand pounds of environmentally released mercury, there was a 61 percent increase in autism rates. The study looked at Texas county-by-county levels of mercury emissions and compared them to the rates of autism and special education services in 1,200 Texas school districts. One county with low mercury emissions but significant autism rates was found to harbor the nation’s largest mercury mine. An author of the study said it shows a potentially “important connection between environmental exposure to mercury and the development of autism.”

61% increase? Funny that, because Professor Palmer himself says

We found that for every 1,000 pounds of mercury released by industry, there was a 17 percent increase in autism,” Palmer tells WebMD.

Please do your research properly. The Professor also says:

Palmer is quick to point out that this kind of study does not prove mercury pollution causes autism. “We show a significant relationship between mercury release into the environment and autism. But that does not prove causation,” Palmer says.

FOX

So, once again. No proof that mercury poisoning either is, or causes, autism. There may be a link but what that link is or even if it exists at all is pure conjecture. In the meantime, what isn’t conjecture is the amount of money Kirby, Generation Rescue et al make from manipulating the emotions of parents into buying hysterical books on the subject and purchasing thousands of dollars worth of chelation ‘therapy’.

I’m merely happy that the more hysterical and easily led of these groups exist mainly in the US and Canada. Thankfully in the UK common sense still seems to largely prevail. Then again, we’ve never been of the opinion that everything difficult can be solved be stamping our feet and showering it with money.